What is Alzheimer’s Disease?

Alzheimer’s disease is an irreversible, progressive brain disease that slowly destroys memory and thinking skills, and eventually even the ability to carry out the simplest tasks. In most people with Alzheimer’s, symptoms first appear after age 60. Estimates vary, but experts suggest that as many as 5.1 million Americans may have Alzheimer’s disease.

Alzheimer’s disease is the most common cause of dementia among older people. Dementia is the loss of cognitive functioning—thinking, remembering, and reasoning—and behavioral abilities, to such an extent that it interferes with a person’s daily life and activities. Dementia ranges in severity from the mildest stage, when it is just beginning to affect a person’s functioning, to the most severe stage, when the person must depend completely on others for basic activities of daily living.

Changes in the Brain in Alzheimer’s Disease

Although we still don’t know how the Alzheimer’s disease process begins, it seems likely that damage to the brain starts a decade or more before problems become evident. During the preclinical stage of Alzheimer’s disease, people are free of symptoms but toxic changes are taking place in the brain. Abnormal deposits of proteins form amyloid plaques and tau tangles throughout the brain, and once-healthy neurons begin to work less efficiently. Over time, neurons lose their ability to function and communicate with each other, and eventually they die.

Before long, the damage spreads to a nearby structure in the brain called the hippocampus, which is essential in forming memories. As more neurons die, affected brain regions begin to shrink. By the final stage of Alzheimer’s, damage is widespread, and brain tissue has shrunk significantly.

Mild Alzheimer’s Disease

As Alzheimer’s disease progresses, memory loss worsens, and changes in other cognitive abilities are evident. Problems can include, for example, getting lost, trouble handling money and paying bills, repeating questions, taking longer to complete normal daily tasks, using poor judgment, and having some mood and personality changes. People often are diagnosed in this stage.

Moderate Alzheimer’s Disease

In this stage, damage occurs in areas of the brain that control language, reasoning, sensory processing, and conscious thought. Memory loss and confusion grow worse, and people begin to have problems recognizing family and friends. They may be unable to learn new things, carry out tasks that involve multiple steps (such as getting dressed), or cope with new situations. They may have hallucinations, delusions, and paranoia, and may behave impulsively.

Severe Alzheimer’s Disease

By the final stage, plaques and tangles have spread throughout the brain, and brain tissue has shrunk significantly. People with severe Alzheimer’s cannot communicate and are completely dependent on others for their care. Near the end, the person may be in bed most or all of the time as the body shuts down.

What Causes Alzheimer’s

Scientists don’t yet fully understand what causes Alzheimer’s disease, but it has become increasingly clear that it develops because of a complex series of events that take place in the brain over a long period of time. It is likely that the causes include some mix of genetic, environmental, and lifestyle factors. Because people differ in their genetic make-up and lifestyle, the importance of any one of these factors in increasing or decreasing the risk of developing Alzheimer’s may differ from person to person.


Early-onset Alzheimer’s is a rare form of the disease. It occurs in people age 30 to 60 and represents less than 5 percent of all people who have Alzheimer’s disease. Most cases of early-onset Alzheimer’s are familial Alzheimer’s disease, caused by changes in one of three known genes inherited from a parent.

Most people with Alzheimer’s disease have “late-onset” Alzheimer’s, which usually develops after age 60. Many studies have linked the apolipoprotein E (APOE) gene to late-onset Alzheimer’s. This gene has several forms. One of them, APOE ε4, seems to increase a person’s risk of getting the disease. However, carrying the APOE ε4 form of the gene does not necessarily mean that a person will develop Alzheimer’s disease, and people carrying no APOE ε4 can also develop the disease.

Most experts believe that additional genes may influence the development of late-onset Alzheimer’s. Scientists around the world are searching for these genes, and have identified a number of common genes in addition to APOE ε4 that may increase a person’s risk for late-onset Alzheimer’s.

Environmental/Lifestyle Factors

Research also suggests that a host of factors beyond basic genetics may play a role in the development and course of Alzheimer’s disease. There is a great deal of interest, for example, in associations between cognitive decline and vascular and metabolic conditions such as heart disease, stroke, high blood pressure, diabetes, and obesity. Understanding these relationships and testing them in clinical trials will help us understand whether reducing risk factors for these conditions may help with Alzheimer’s as well.

Further, a nutritious diet, physical activity, social engagement, and mentally stimulating pursuits can all help people stay healthy as they age. New research suggests the possibility that these and other factors also might help to reduce the risk of cognitive decline and Alzheimer’s disease. Clinical trials of specific interventions are underway to test some of these possibilities.

NOTE: Information provided by the National Institutes of Health

NIH…Turning Discovery Into Health®; U.S. Department of Health and Human Services; Publication Date: September 2012; Page Last Updated: August 27, 2014

Brain Tissue Cells Development Proposal

 The overall purpose of this project is to reverse damaged brain tissue memory and loss of cells due to Alzheimer.  Additional focus will be to conversely develop and/or re-establishes intellectual/social skills. These skills are cognitive, mental, and processes which are related to knowledge, attention, reasoning, memory/working memory, judgment, evaluation, computation, problem solving, decision making, comprehension and the production of language.  Also, we believe the same group of brain tissue-cells disorder results in the lack of mental functions with children who suffer from Autism.

Alzheimer’s disease is a progressive disease that destroys memory and other important mental functions. The brain cells themselves degenerate and die, causing a steady decline in memory, and social/intellectual skills. These changes are severe enough to interfere with day-to-day life which affects 36 million people worldwide and unfortunately, there is no cure.

The hypothesis is: Specific blended/measured all-natural ingredients from the land and sea reverses or prevents genetic and environmental (diet, injury, pollution, surgery, bacteria, injections, pesticides, etc.) factors that causes ‘Brain Tissue-cells’ to malfunction. This creates an imbalance of chemicals and the lack of immune system instruction to cleanse the brain – – the body.  Furthermore, when similar cells are destroyed or cannot perform a common function the communication from cell-to-cell is degraded disrupting nutrients, blood and oxygen from flowing to the brain as well as preventing waste (toxin, water, and carbon-dioxide) from being remove from the brain.  Therefore, genetic and environmental conditions affect normal brain tissue cells and the immune system from a natural biological process of healing (cleansing) tissues in the brain – -and other tissue-cells within the body. This process holds true for all the systems within the human body; muscular, nervous, digestive system, cardiovascular, etc.

How it works:

We believe at TiMae Health, Alzheimer is caused by viruses, external forces, diet, and the byproduct
effects are as follows:
• Beta-amyloid plaques – sticky clumps of protein fragments and cellular material
• Neurofibrillary tangles – insoluble twisted fibers composed largely of the protein tau that build up
• Connections lost – Alzheimer interferes with neurons/neurotransmitters (cell signal transmission)
• Brain inflammation – overactive Microglia fighting Beta-amyloid plaques
• Brain shrinks – cortex & hippocampus are affected
• Brain cells death


Kills the virus, prevent further Beta-amyloid plaques from forming, correct-tangles, reduce
inflammation, remove waste, and replenishes tissue-cells with nutrients.


1. Halts the progression of the disease
2. Allows the free flow of water, blood, oxygen and nutrients throughout the nervous system
3. Reestablishes connection between the cells – Synapses (Information passes between nerve cells)
4. Waste removal
5. Preventing brain cells death
6. Gradual process of cognitive skills resurgent/development
7. Living longer and in comfort

Next Step I: Brain cognitive development skills — expenses include travel to Asia Pacific for ingredients. The next sequential step in the process is to obtain patent, and national certification to show product safety/nutritional facts as well show ‘NO’ performance enhancing chemicals.


Test evaluation: For memory loss – 15 control questions daily; for cognitive skills – assembling puzzles, comprehension, writing/drawing an object/person, understanding pictures/books and sitting/watching TV. Document results and continue to add decision-making activities daily.


This statement has not been evaluated by the FDA. This product is not intended to diagnose, treat, cure, or prevent any disease”?